Quote:
Originally Posted by amRam
What's the point of telling this to laymen when you know that there is a pathway for consumed fats to end up as glucose via glycerol gluconeogenesis?
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That pathway is only used if you're in real trouble (freezing to death, for example), and it involves lactate and AA's much more than glycerol. You're nit-picking at something I suspect you googled, and are trying to invite me to a gun-fight while you're holding a paperclip.
In general, dietary proteins contribute little to glucose production, and fats, almost none. 6th grade explanation: In your liver mostly, lipase breaks fats into fatty acids, which are moved by blood into the mitochondria of skeletal muscle, and yield ATP, via citric acid cycle. Fat can be an excellent source of energy (as you're sitting on your computer chair reading this, about 80% of the energy you are using is coming from fat), and oxidation of fatty acids can become more efficient with training. Long periods of aerobic exercise with little carbohydrate consumption deplete glycogen stores (in the liver and sarcolemma), and force increased production of lipase (liver and other places, but mostly liver), and acetyl coenzyme A in the mitochondria. Over months of this, mitochondrial density can be increased.
For juggernaut: If you not eat lot food, and exercise slow and long time anyway, you will get gooder at picking fat for teh go-go juice.